Adrianalyn

Adrianalyn


Similarly, because of its extreme sensitivity, much ischemia research has focused on the hippocampus; however the mechanisms gleaned from the hippocampus may not extrapolate to other brain regions, especially those with fundamentally different cellular and molecular make-ups like the cerebellum. The mechanisms underlying early responses to ischemia were also investigated. Brain ischemia triggers a sequence of events, which can be classified in two phases: Evoked responses in CA1 neurons induced by stimulation of CA3 axons are reduced by actin filament stabilization, due to a decrease in the probability of release. This difference in the ischemic response could be explained by intrinsic differences in the synaptic input to each of these cells.

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Adrianalyn


Similarly, because of its extreme sensitivity, much ischemia research has focused on the hippocampus; however the mechanisms gleaned from the hippocampus may not extrapolate to other brain regions, especially those with fundamentally different cellular and molecular make-ups like the cerebellum. The mechanisms underlying early responses to ischemia were also investigated. Brain ischemia triggers a sequence of events, which can be classified in two phases: Evoked responses in CA1 neurons induced by stimulation of CA3 axons are reduced by actin filament stabilization, due to a decrease in the probability of release. This difference in the ischemic response could be explained by intrinsic differences in the synaptic input to each of these cells. Adrianalyn

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  1. Thus, the early and late ischemic responses vary in different brain regions and this thesis has covered several gaps in the existing knowledge in the ischemia field. Similarly, because of its extreme sensitivity, much ischemia research has focused on the hippocampus; however the mechanisms gleaned from the hippocampus may not extrapolate to other brain regions, especially those with fundamentally different cellular and molecular make-ups like the cerebellum.

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